UV-C absorption in Thermal/UV Keratopathy can hyperactivate which ion channels?

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Prepare for the NBEO Ocular Disease Part 1 Test. Study with multiple-choice questions and detailed explanations. Enhance your test readiness!

Multiple Choice

UV-C absorption in Thermal/UV Keratopathy can hyperactivate which ion channels?

Explanation:
UV-C–induced photokeratitis triggers cellular stress that raises intracellular calcium in corneal epithelial cells. That increase in Ca2+ activates calcium-activated potassium channels, causing potassium to efflux from the cell. The outward K+ current hyperpolarizes the cell membrane and is a prominent ionic response to the UV-C injury in Thermal/UV keratopathy. So, the ion channels most notably hyperactivated in this context are potassium channels.

UV-C–induced photokeratitis triggers cellular stress that raises intracellular calcium in corneal epithelial cells. That increase in Ca2+ activates calcium-activated potassium channels, causing potassium to efflux from the cell. The outward K+ current hyperpolarizes the cell membrane and is a prominent ionic response to the UV-C injury in Thermal/UV keratopathy. So, the ion channels most notably hyperactivated in this context are potassium channels.

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